Neurological diseases >>>> Guillain-Barre syndrome
Guillain-Barre syndrome.
Guillain-Barre syndrome or acute post-infectious polyneuropathy is an autoimmune disease that manifests itself in the development of acute paresis and paralysis of peripheral nerves, cranial and spinal nerve trunks, disrupting the motor activity of the articular-muscular apparatus, muscle tissue sensitivity and tendon reflexes. Most often, the muscles responsible for breathing and swallowing are involved in the process of the disease, which makes the course of the disease extremely difficult.
Autoimmune processes aimed at destroying the myelin of neurons and lemmocytes (auxiliary nerve cells located along peripheral axons and performing a supporting and trophic function) are provoked by acute infectious diseases (respiratory or intestinal infections of bacterial and viral origin). Sometimes the impetus for the development of the syndrome is given by trauma of peripheral nerves, hypothermia, or the disease develops as a complication of surgery, vaccination.
Increasing muscle paresis (muscle weakness) and loss of tendon reflexes are considered typical signs of Guillain-Barre syndrome.
Despite the severe course of the disease, a person with Guillain-Barre syndrome has every chance of being fully rehabilitated through a course of treatment with immunoglobulins (IgG class), plasmapheresis sessions (4-5 sessions) and a timely long-term connection to a ventilator. It is possible to independently recover neurons for a long time (about six months) against the background of mandatory connection to the mechanical ventilation equipment for the entire rehabilitation period. According to statistics, the deaths of the disease are caused by the insufficient preparedness of medical institutions to serve patients with this type of disorder.
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